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Researcher · Cégep de Jonquière · Canada

Benjamin Gallais

Benjamin Gallais publishes mostly in Cellular and Molecular Neuroscience, Neurology and Molecular Biology, on topics such as Genetic Neurodegenerative Diseases, Parkinson's Disease Mechanisms and Treatments and Amyotrophic Lateral Sclerosis Research.

ORCID 0000-0003-0273-8298 OpenAlex
World rank
#2,958,035
of 7,710,434 ranked researchers
Rank in Canada
#95,317
of 183,316
Works
65
Citations
811
Citations per work
12.5
h-index
13
i10-index 17Top 62% in Cellular and Molecular Neuroscience
Benjamin Gallais

What does Benjamin Gallais research?

Benjamin Gallais's work is mostly in Neuroscience (23%), Medicine (20%), Biochemistry, Genetics and Molecular Biology (9%).

Everything else: 23 works · 36.0% of their workLife Sciences: 21 works · 32.3% of their workNeuroscience: 15 works · 23.2% of their workCellular and Molecular Neuroscience: 15 works · 23.2% of their workBiochemistry, Genetics and Molecular Biology: 6 works · 9.1% of their workMolecular Biology: 6 works · 9.1% of their workHealth Sciences: 17 works · 26.2% of their workMedicine: 13 works · 20.1% of their workNeurology: 9 works · 14.0% of their workPsychiatry and Mental health: 4 works · 6.1% of their workHealth Professions: 4 works · 6.1% of their workGeneral Health Professions: 4 works · 6.1% of their workSocial Sciences: 4 works · 5.5% of their workSocial Sciences: 4 works · 5.5% of their workSociology and Political Science: 4 works · 5.5% of their workTHEIR WORKBenjamin Gallais65works
Life Sciences32%Health Sciences26%Social Sciences6%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
  1. Everything else2336.0%
    1. Neuroscience1523.2%
    2. Biochemistry, Genetics and Molecular Biology69.1%
    1. Medicine1320.1%
    2. Health Professions46.1%
    1. Social Sciences45.5%
  1. 1Cellular and Molecular Neuroscience23% of their works
  2. 2Neurology14% of their works
  3. 3Molecular Biology9% of their works
  4. 4General Health Professions6% of their works
  5. 5Psychiatry and Mental health6% of their works
  6. 6Sociology and Political Science6% of their works

Research topics

Which keywords describe Benjamin Gallais's research?

The keywords of their largest research topics: Mitochondrial Dysfunction, Huntington's Disease, Protein Aggregation, Neurodegeneration, Diagnostic Criteria, Oxidative Stress, a-Synuclein and Parkinson's Disease.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most.

All 20 words, with their numbers
  1. Mitochondrial Dysfunction483 topics
  2. Huntington's Disease331 topic
  3. Protein Aggregation331 topic
  4. Neurodegeneration172 topics
  5. Diagnostic Criteria162 topics
  6. Oxidative Stress152 topics
  7. Parkinson's Disease101 topic
  8. a-Synuclein101 topic
  9. Cultural Intelligence71 topic
  10. FUS/TLS71 topic
  11. Intercultural Competence71 topic
  12. TDP-4371 topic
  13. Chronic Fatigue Syndrome61 topic
  14. Fibromyalgia61 topic
  15. Muscle Regeneration61 topic
  16. Skeletal Muscle Atrophy61 topic
  17. Healthcare Legislation51 topic
  18. Mitochondria51 topic
  19. Patient Rights51 topic
  20. Reactive Oxygen Species51 topic

Where does Benjamin Gallais work?

Benjamin Gallais's main affiliation in the publication record is Cégep de Jonquière, Canada.

How many publications and citations does Benjamin Gallais have?

OpenAlex counts 65 works and 811 citations for Benjamin Gallais, an h-index of 13, ranking #2,958,035 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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