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Researcher · CHDI Foundation · United States

Celia Dominguez

Celia Dominguez publishes mostly in Molecular Biology, Cellular and Molecular Neuroscience and Organic Chemistry, on topics such as Genetic Neurodegenerative Diseases, Mitochondrial Function and Pathology and Neurological disorders and treatments.

ORCID 0000-0001-6862-1645 OpenAlex
World rank
#1,623,457
of 7,710,434 ranked researchers
Rank in United States
#504,397
of 1,627,098
Works
145
Citations
3.8k
Citations per work
26.3
h-index
37
i10-index 81Top 8.9% in Molecular Biology
Celia Dominguez

What does Celia Dominguez research?

Celia Dominguez's work is mostly in Biochemistry, Genetics and Molecular Biology (30%), Neuroscience (17%), Chemistry (17%).

Life Sciences: 68 works · 47.1% of their workBiochemistry, Genetics and Molecular Biology: 44 works · 30.0% of their workMolecular Biology: 44 works · 30.0% of their workNeuroscience: 25 works · 17.1% of their workCellular and Molecular Neuroscience: 25 works · 17.1% of their workEverything else: 37 works · 25.6% of their workPhysical Sciences: 24 works · 16.6% of their workChemistry: 24 works · 16.6% of their workOrganic Chemistry: 24 works · 16.6% of their workHealth Sciences: 16 works · 10.7% of their workMedicine: 16 works · 10.7% of their workNeurology: 9 works · 6.2% of their workRadiology, Nuclear Medicine and Imaging: 4 works · 2.5% of their workOncology: 3 works · 2.0% of their workTHEIR WORKCelia Dominguez145works
Life Sciences47%Physical Sciences17%Health Sciences11%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology4430.0%
    2. Neuroscience2517.1%
  2. Everything else3725.6%
    1. Chemistry2416.6%
    1. Medicine1610.7%
  1. 1Molecular Biology30% of their works
  2. 2Cellular and Molecular Neuroscience17% of their works
  3. 3Organic Chemistry17% of their works
  4. 4Neurology6% of their works
  5. 5Radiology, Nuclear Medicine and Imaging2% of their works
  6. 6Oncology2% of their works

Research topics

Which keywords describe Celia Dominguez's research?

The keywords of their largest research topics: Mitochondrial Dysfunction, Huntington's Disease, Protein Aggregation, Neurological Disorders, Mitochondria, Reactive Oxygen Species, Deep Brain Stimulation and Parkinson's Disease.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 18 words, with their numbers
  1. Mitochondrial Dysfunction772 topics
  2. Huntington's Disease531 topic
  3. Protein Aggregation531 topic
  4. Neurological Disorders282 topics
  5. Mitochondria241 topic
  6. Reactive Oxygen Species241 topic
  7. Deep Brain Stimulation201 topic
  8. Parkinson's Disease201 topic
  9. Drug Discovery131 topic
  10. Peptide Synthesis131 topic
  11. Quinoxaline121 topic
  12. Synthesis121 topic
  13. Neurotransmission81 topic
  14. Synaptic Plasticity81 topic
  15. Alternative Splicing71 topic
  16. BRAF gene71 topic
  17. MAP Kinase71 topic
  18. RNA-Binding Proteins71 topic

Where does Celia Dominguez work?

Celia Dominguez's main affiliation in the publication record is CHDI Foundation, United States.

How many publications and citations does Celia Dominguez have?

OpenAlex counts 145 works and 3,818 citations for Celia Dominguez, an h-index of 37, ranking #1,623,457 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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