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Researcher · CHDI Foundation · United States

David Howland

David Howland publishes mostly in Molecular Biology, Cellular and Molecular Neuroscience and Neurology, on topics such as Genetic Neurodegenerative Diseases, Mitochondrial Function and Pathology and Muscle Physiology and Disorders.

OpenAlex
World rank
#533,136
of 7,710,434 ranked researchers
Rank in United States
#184,726
of 1,627,098
Works
101
Citations
7.3k
Citations per work
72.7
h-index
39
i10-index 63Top 7.8% in Molecular Biology
David Howland

What does David Howland research?

David Howland's work is mostly in Biochemistry, Genetics and Molecular Biology (39%), Neuroscience (29%), Medicine (14%).

Life Sciences: 69 works · 68.3% of their workBiochemistry, Genetics and Molecular Biology: 40 works · 39.5% of their workMolecular Biology: 37 works · 36.9% of their workCell Biology: 3 works · 2.6% of their workNeuroscience: 29 works · 28.8% of their workCellular and Molecular Neuroscience: 29 works · 28.8% of their workHealth Sciences: 17 works · 16.8% of their workMedicine: 14 works · 14.2% of their workNeurology: 11 works · 10.6% of their workPhysiology: 4 works · 3.6% of their workHealth Professions: 3 works · 2.6% of their workGeneral Health Professions: 3 works · 2.6% of their workEverything else: 15 works · 14.9% of their workTHEIR WORKDavid Howland101works
Life Sciences68%Health Sciences17%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology4039.5%
    2. Neuroscience2928.8%
    1. Medicine1414.2%
    2. Health Professions32.6%
  3. Everything else1514.9%
  1. 1Molecular Biology37% of their works
  2. 2Cellular and Molecular Neuroscience29% of their works
  3. 3Neurology11% of their works
  4. 4Physiology4% of their works
  5. 5General Health Professions3% of their works
  6. 6Cell Biology3% of their works

Research topics

Which keywords describe David Howland's research?

The keywords of their largest research topics: Mitochondrial Dysfunction, Protein Aggregation, Huntington's Disease, Polyglutamine, Oxidative Stress, Neurodegenerative Diseases, Mitochondria and Reactive Oxygen Species.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 21 words, with their numbers
  1. Mitochondrial Dysfunction972 topics
  2. Protein Aggregation692 topics
  3. Huntington's Disease631 topic
  4. Polyglutamine631 topic
  5. Oxidative Stress412 topics
  6. Neurodegenerative Diseases402 topics
  7. Mitochondria341 topic
  8. Reactive Oxygen Species341 topic
  9. Muscle Regeneration231 topic
  10. Skeletal Muscle Atrophy231 topic
  11. Neurodegeneration202 topics
  12. FUS/TLS131 topic
  13. TDP-43131 topic
  14. Deep Brain Stimulation111 topic
  15. Parkinson's Disease111 topic
  16. Amyloid Hypothesis71 topic
  17. Tau Pathology71 topic
  18. Molecular Basis61 topic
  19. Prion Diseases61 topic
  20. Alternative Splicing51 topic
  21. RNA-Binding Proteins51 topic

Where does David Howland work?

David Howland's main affiliation in the publication record is CHDI Foundation, United States.

How many publications and citations does David Howland have?

OpenAlex counts 101 works and 7,345 citations for David Howland, an h-index of 39, ranking #533,136 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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