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Researcher · Aarhus Municipality · Denmark

Eigil Iversen

Eigil Iversen publishes mostly in Endocrinology, Diabetes and Metabolism, Cellular and Molecular Neuroscience and Physiology, on topics such as Thyroid Disorders and Treatments, Neuropeptides and Animal Physiology and Growth Hormone and Insulin-like Growth Factors.

OpenAlex
World rank
#3,457,886
of 7,710,434 ranked researchers
Rank in Denmark
#25,267
of 43,819
Works
18
Citations
771
Citations per work
42.8
h-index
9
i10-index 9Top 63% in Endocrinology, Diabetes and Metabolism
Eigil Iversen

What does Eigil Iversen research?

Eigil Iversen's work is mostly in Medicine (61%), Neuroscience (14%), Biochemistry, Genetics and Molecular Biology (6%).

Health Sciences: 11 works · 61.2% of their workMedicine: 11 works · 61.2% of their workEndocrinology, Diabetes and Metabolism: 10 works · 52.8% of their workPhysiology: 1 works · 5.6% of their workPediatrics, Perinatology and Child Health: 1 works · 2.8% of their workLife Sciences: 4 works · 19.5% of their workNeuroscience: 3 works · 13.9% of their workCellular and Molecular Neuroscience: 3 works · 13.9% of their workBiochemistry, Genetics and Molecular Biology: 1 works · 5.6% of their workMolecular Biology: 1 works · 5.6% of their workEverything else: 3 works · 16.5% of their workSocial Sciences: 1 works · 2.8% of their workSocial Sciences: 1 works · 2.8% of their workPolitical Science and International Relations: 1 works · 2.8% of their workTHEIR WORKEigil Iversen18works
Life Sciences20%Health Sciences61%Social Sciences3%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Medicine1161.2%
    1. Neuroscience313.9%
    2. Biochemistry, Genetics and Molecular Biology15.6%
  3. Everything else316.5%
    1. Social Sciences12.8%
  1. 1Endocrinology, Diabetes and Metabolism53% of their works
  2. 2Cellular and Molecular Neuroscience14% of their works
  3. 3Physiology6% of their works
  4. 4Molecular Biology6% of their works
  5. 5Political Science and International Relations3% of their works
  6. 6Pediatrics, Perinatology and Child Health3% of their works

Research topics

Which keywords describe Eigil Iversen's research?

The keywords of their largest research topics: Cardiovascular Risk, Thyroid Dysfunction, Thyroid Hormone, IGF-1, Insulin-like Growth Factors, Neuropeptides, Receptors and Cell Signaling.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 18 words, with their numbers
  1. Cardiovascular Risk132 topics
  2. Thyroid Dysfunction111 topic
  3. Thyroid Hormone111 topic
  4. IGF-141 topic
  5. Insulin-like Growth Factors41 topic
  6. Neuropeptides41 topic
  7. Receptors41 topic
  8. Cell Signaling22 topics
  9. Differentiated Thyroid Cancer21 topic
  10. Hormone Therapy21 topic
  11. Testosterone21 topic
  12. Thyroid Nodules21 topic
  13. Crystal Structure11 topic
  14. G-Protein-Coupled Receptors11 topic
  15. Ion Channels11 topic
  16. Neurotransmission11 topic
  17. Synaptic Plasticity11 topic
  18. Voltage-Gated Channels11 topic

Where does Eigil Iversen work?

Eigil Iversen's main affiliation in the publication record is Aarhus Municipality, Denmark.

How many publications and citations does Eigil Iversen have?

OpenAlex counts 18 works and 771 citations for Eigil Iversen, an h-index of 9, ranking #3,457,886 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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