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Researcher · Fundación Instituto Leloir · Argentina

Fernando Juan Pitossi

Fernando Juan Pitossi publishes mostly in Neurology, Molecular Biology and Cellular and Molecular Neuroscience, on topics such as Neuroinflammation and Neurodegeneration Mechanisms, Neurogenesis and neuroplasticity mechanisms and Nerve injury and regeneration.

ORCID 0000-0001-5104-9726 OpenAlex
World rank
#691,300
of 7,710,434 ranked researchers
Rank in Argentina
#848
of 38,473
Works
105
Citations
7.4k
Citations per work
70.5
h-index
39
i10-index 61Top 4.8% in Neurology (Neuroscience)
Fernando Juan Pitossi

What does Fernando Juan Pitossi research?

Fernando Juan Pitossi's work is mostly in Neuroscience (39%), Biochemistry, Genetics and Molecular Biology (16%), Immunology and Microbiology (9%).

Life Sciences: 69 works · 65.3% of their workNeuroscience: 41 works · 39.5% of their workNeurology: 19 works · 18.0% of their workCellular and Molecular Neuroscience: 13 works · 12.5% of their workDevelopmental Neuroscience: 9 works · 9.0% of their workBiochemistry, Genetics and Molecular Biology: 17 works · 16.4% of their workMolecular Biology: 17 works · 16.4% of their workImmunology and Microbiology: 10 works · 9.4% of their workImmunology: 10 works · 9.4% of their workEverything else: 32 works · 30.4% of their workHealth Sciences: 5 works · 4.3% of their workMedicine: 5 works · 4.3% of their workPhysiology: 5 works · 4.3% of their workTHEIR WORKFernando JuanPitossi105works
Life Sciences65%Health Sciences4%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Neuroscience4139.5%
    2. Biochemistry, Genetics and Molecular Biology1716.4%
    3. Immunology and Microbiology109.4%
  2. Everything else3230.4%
    1. Medicine54.3%
  1. 1Neurology18% of their works
  2. 2Molecular Biology16% of their works
  3. 3Cellular and Molecular Neuroscience12% of their works
  4. 4Immunology9% of their works
  5. 5Developmental Neuroscience9% of their works
  6. 6Physiology4% of their works

Research topics

Which keywords describe Fernando Juan Pitossi's research?

The keywords of their largest research topics: Brain Development, Neurodegeneration, Microglia, Neuroinflammation, Synaptic Plasticity, Depression, Adult Stem Cells and Neurogenesis.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 20 words, with their numbers
  1. Brain Development592 topics
  2. Neurodegeneration462 topics
  3. Microglia391 topic
  4. Neuroinflammation391 topic
  5. Synaptic Plasticity222 topics
  6. Depression212 topics
  7. Adult Stem Cells201 topic
  8. Neurogenesis201 topic
  9. BDNF141 topic
  10. Neurotrophins141 topic
  11. Induced Pluripotent Stem Cells101 topic
  12. Pattern Recognition Receptors101 topic
  13. Pluripotent Stem Cells101 topic
  14. Toll-like Receptors101 topic
  15. GABAergic81 topic
  16. Neurotransmission81 topic
  17. Brain71 topic
  18. Parkinson's Disease71 topic
  19. Stress71 topic
  20. a-Synuclein71 topic

Where does Fernando Juan Pitossi work?

Fernando Juan Pitossi's main affiliation in the publication record is Fundación Instituto Leloir, Argentina.

How many publications and citations does Fernando Juan Pitossi have?

OpenAlex counts 105 works and 7,404 citations for Fernando Juan Pitossi, an h-index of 39, ranking #691,300 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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