Science Explorer Back to mapMap
Discussion
Researcher · Fukuoka University · Japan

Hitomi Nishinakamura

Hitomi Nishinakamura publishes mostly in Oncology, Molecular Biology and Immunology, on topics such as Pancreatic function and diabetes, Cancer Immunotherapy and Biomarkers and Diabetes and associated disorders.

ORCID 0009-0008-4604-5796 OpenAlex
World rank
#822,337
of 7,710,434 ranked researchers
Rank in Japan
#10,224
of 383,136
Works
36
Citations
3.4k
Citations per work
94.2
h-index
16
i10-index 19Top 33% in Oncology
Hitomi Nishinakamura

What does Hitomi Nishinakamura research?

Hitomi Nishinakamura's work is mostly in Medicine (36%), Biochemistry, Genetics and Molecular Biology (23%), Immunology and Microbiology (15%).

Life Sciences: 14 works · 38.0% of their workBiochemistry, Genetics and Molecular Biology: 8 works · 22.8% of their workMolecular Biology: 5 works · 15.2% of their workGenetics: 3 works · 7.6% of their workImmunology and Microbiology: 5 works · 15.2% of their workImmunology: 5 works · 15.2% of their workHealth Sciences: 13 works · 36.2% of their workMedicine: 13 works · 36.2% of their workOncology: 7 works · 18.1% of their workSurgery: 4 works · 12.4% of their workEndocrinology, Diabetes and Metabolism: 2 works · 5.7% of their workEverything else: 9 works · 25.8% of their workTHEIR WORKHitomiNishinakamura36works
Life Sciences38%Health Sciences36%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology822.8%
    2. Immunology and Microbiology515.2%
    1. Medicine1336.2%
  3. Everything else925.8%
  1. 1Oncology18% of their works
  2. 2Molecular Biology15% of their works
  3. 3Immunology15% of their works
  4. 4Surgery12% of their works
  5. 5Genetics8% of their works
  6. 6Endocrinology, Diabetes and Metabolism6% of their works

Research topics

Which keywords describe Hitomi Nishinakamura's research?

The keywords of their largest research topics: Tumor Microenvironment, Pancreatic Islets, ß-Cell Dysfunction, Inflammation, Immune Checkpoint Blockade, PD-1 and PD-L1, Genetics and Natural Killer Cells.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 18 words, with their numbers
  1. Tumor Microenvironment112 topics
  2. Pancreatic Islets91 topic
  3. ß-Cell Dysfunction91 topic
  4. Inflammation82 topics
  5. Immune Checkpoint Blockade71 topic
  6. PD-1 and PD-L171 topic
  7. Genetics61 topic
  8. NK Cell Recognition61 topic
  9. Natural Killer Cells61 topic
  10. Type 1 Diabetes61 topic
  11. IL-641 topic
  12. Lymphangiogenesis41 topic
  13. STAT341 topic
  14. VEGF41 topic
  15. VEGF-C41 topic
  16. endothelial progenitor cells41 topic
  17. Alternative Splicing31 topic
  18. RNA-Binding Proteins31 topic

Where does Hitomi Nishinakamura work?

Hitomi Nishinakamura's main affiliation in the publication record is Fukuoka University, Japan.

How many publications and citations does Hitomi Nishinakamura have?

OpenAlex counts 36 works and 3,390 citations for Hitomi Nishinakamura, an h-index of 16, ranking #822,337 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

Ask a question or share a note about Hitomi Nishinakamura. Keep it to the research: no personal attacks, advertising or links. Any reader can report a post, which collapses it for everyone until it is opened.