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Researcher · University of Southern Denmark · Denmark

Kate Lykke Lambertsen

Kate Lykke Lambertsen publishes mostly in Neurology, Immunology and Molecular Biology, on topics such as Neuroinflammation and Neurodegeneration Mechanisms, Neurological Disease Mechanisms and Treatments and Immune Response and Inflammation.

ORCID 0000-0001-9276-4902 OpenAlex
World rank
#568,254
of 7,710,434 ranked researchers
Rank in Denmark
#4,233
of 43,819
Works
204
Citations
6.7k
Citations per work
32.8
h-index
41
i10-index 95Top 4.2% in Neurology (Neuroscience)
Kate Lykke Lambertsen

What does Kate Lykke Lambertsen research?

Kate Lykke Lambertsen's work is mostly in Neuroscience (30%), Immunology and Microbiology (12%), Medicine (10%).

Life Sciences: 103 works · 50.7% of their workNeuroscience: 62 works · 30.2% of their workNeurology: 52 works · 25.6% of their workCellular and Molecular Neuroscience: 9 works · 4.6% of their workImmunology and Microbiology: 24 works · 11.6% of their workImmunology: 24 works · 11.6% of their workBiochemistry, Genetics and Molecular Biology: 18 works · 8.9% of their workMolecular Biology: 18 works · 8.9% of their workEverything else: 79 works · 38.8% of their workHealth Sciences: 21 works · 10.5% of their workMedicine: 21 works · 10.5% of their workSurgery: 13 works · 6.2% of their workPathology and Forensic Medicine: 9 works · 4.3% of their workTHEIR WORKKate LykkeLambertsen204works
Life Sciences51%Health Sciences10%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Neuroscience6230.2%
    2. Immunology and Microbiology2411.6%
    3. Biochemistry, Genetics and Molecular Biology188.9%
  2. Everything else7938.8%
    1. Medicine2110.5%
  1. 1Neurology26% of their works
  2. 2Immunology12% of their works
  3. 3Molecular Biology9% of their works
  4. 4Surgery6% of their works
  5. 5Cellular and Molecular Neuroscience5% of their works
  6. 6Pathology and Forensic Medicine4% of their works

Research topics

Which keywords describe Kate Lykke Lambertsen's research?

The keywords of their largest research topics: Brain Development, Ischemic Stroke, Microglia, Neuroinflammation, Inflammation, Cerebral Hypoperfusion, Vascular Cognitive Impairment and Pattern Recognition Receptors.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 19 words, with their numbers
  1. Brain Development1142 topics
  2. Ischemic Stroke1132 topics
  3. Microglia981 topic
  4. Neuroinflammation981 topic
  5. Inflammation442 topics
  6. Cerebral Hypoperfusion301 topic
  7. Vascular Cognitive Impairment301 topic
  8. Pattern Recognition Receptors251 topic
  9. Toll-like Receptors251 topic
  10. Activation191 topic
  11. Macrophage191 topic
  12. Adult Stem Cells161 topic
  13. Neurogenesis161 topic
  14. Endovascular Therapy151 topic
  15. Neurotransmission151 topic
  16. Synaptic Plasticity151 topic
  17. Thrombolysis151 topic
  18. Blood-Brain Barrier121 topic
  19. Neurovascular Unit121 topic

Where does Kate Lykke Lambertsen work?

Kate Lykke Lambertsen's main affiliation in the publication record is University of Southern Denmark, Denmark.

How many publications and citations does Kate Lykke Lambertsen have?

OpenAlex counts 204 works and 6,691 citations for Kate Lykke Lambertsen, an h-index of 41, ranking #568,254 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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