Science Explorer Back to mapMap
Discussion
Researcher · Central Institute for Experimental Animals · Japan

Kimio Kobayashi

Kimio Kobayashi publishes mostly in Molecular Biology, Immunology and Genetics, on topics such as Immune Cell Function and Interaction, Hematopoietic Stem Cell Transplantation and T-cell and B-cell Immunology.

ORCID 0000-0003-4232-6204 OpenAlex
World rank
#1,387,886
of 7,710,434 ranked researchers
Rank in Japan
#21,417
of 383,136
Works
70
Citations
4.4k
Citations per work
63.0
h-index
25
i10-index 36Top 22% in Molecular Biology
Kimio Kobayashi

What does Kimio Kobayashi research?

Kimio Kobayashi's work is mostly in Biochemistry, Genetics and Molecular Biology (33%), Medicine (14%), Immunology and Microbiology (14%).

Life Sciences: 33 works · 47.0% of their workBiochemistry, Genetics and Molecular Biology: 23 works · 33.2% of their workMolecular Biology: 17 works · 23.8% of their workGenetics: 7 works · 9.4% of their workImmunology and Microbiology: 10 works · 13.8% of their workImmunology: 10 works · 13.8% of their workEverything else: 27 works · 38.5% of their workHealth Sciences: 10 works · 14.5% of their workMedicine: 10 works · 14.5% of their workHematology: 4 works · 6.3% of their workPhysiology: 3 works · 4.4% of their workOncology: 3 works · 3.8% of their workTHEIR WORKKimio Kobayashi70works
Life Sciences47%Health Sciences14%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology2333.2%
    2. Immunology and Microbiology1013.8%
  2. Everything else2738.5%
    1. Medicine1014.5%
  1. 1Molecular Biology24% of their works
  2. 2Immunology14% of their works
  3. 3Genetics9% of their works
  4. 4Hematology6% of their works
  5. 5Physiology4% of their works
  6. 6Oncology4% of their works

Research topics

Which keywords describe Kimio Kobayashi's research?

The keywords of their largest research topics: Metabolic Syndrome, Genomic Integration, Natural Killer Cells, NK Cell Recognition, Bone Marrow Niche, Hematopoietic Stem Cells, Foxp3 and Gestational Diabetes Mellitus.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 18 words, with their numbers
  1. Metabolic Syndrome112 topics
  2. Genomic Integration92 topics
  3. NK Cell Recognition91 topic
  4. Natural Killer Cells91 topic
  5. Bone Marrow Niche71 topic
  6. Hematopoietic Stem Cells71 topic
  7. Foxp361 topic
  8. Gestational Diabetes Mellitus61 topic
  9. Maternal Obesity61 topic
  10. TGF-ß61 topic
  11. Early-Life Conditions51 topic
  12. Fetal Programming51 topic
  13. Genetic Modification51 topic
  14. Genome Engineering51 topic
  15. RNA-Guided51 topic
  16. Transgenic Animals51 topic
  17. Gene Therapy41 topic
  18. Viral Vectors41 topic

Where does Kimio Kobayashi work?

Kimio Kobayashi's main affiliation in the publication record is Central Institute for Experimental Animals, Japan.

How many publications and citations does Kimio Kobayashi have?

OpenAlex counts 70 works and 4,410 citations for Kimio Kobayashi, an h-index of 25, ranking #1,387,886 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

Ask a question or share a note about Kimio Kobayashi. Keep it to the research: no personal attacks, advertising or links. Any reader can report a post, which collapses it for everyone until it is opened.