Science Explorer Back to mapMap
Discussion
Researcher · Rockefeller University · United States

Masato Ogishi

Masato Ogishi publishes mostly in Immunology, Molecular Biology and Epidemiology, on topics such as Immunodeficiency and Autoimmune Disorders, Immune Cell Function and Interaction and T-cell and B-cell Immunology.

ORCID 0000-0003-2421-7389 OpenAlex
World rank
#66,929
of 7,710,434 ranked researchers
Rank in United States
#25,807
of 1,627,098
Works
88
Citations
8.4k
Citations per work
95.4
h-index
29
i10-index 47Top 16% in Immunology
Masato Ogishi

What does Masato Ogishi research?

Masato Ogishi's work is mostly in Immunology and Microbiology (42%), Biochemistry, Genetics and Molecular Biology (17%), Medicine (15%).

Life Sciences: 52 works · 59.4% of their workImmunology and Microbiology: 37 works · 42.2% of their workImmunology: 37 works · 42.2% of their workBiochemistry, Genetics and Molecular Biology: 15 works · 17.2% of their workMolecular Biology: 10 works · 11.1% of their workGenetics: 5 works · 6.1% of their workEverything else: 22 works · 25.4% of their workHealth Sciences: 13 works · 15.2% of their workMedicine: 13 works · 15.2% of their workEpidemiology: 6 works · 6.6% of their workInfectious Diseases: 4 works · 4.9% of their workOncology: 3 works · 3.7% of their workTHEIR WORKMasato Ogishi88works
Life Sciences59%Health Sciences15%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Immunology and Microbiology3742.2%
    2. Biochemistry, Genetics and Molecular Biology1517.2%
  2. Everything else2225.4%
    1. Medicine1315.2%
  1. 1Immunology42% of their works
  2. 2Molecular Biology11% of their works
  3. 3Epidemiology7% of their works
  4. 4Genetics6% of their works
  5. 5Infectious Diseases5% of their works
  6. 6Oncology4% of their works

Research topics

Which keywords describe Masato Ogishi's research?

The keywords of their largest research topics: Autoimmunity, Genetic Mutations, Primary Immunodeficiency, Foxp3, Natural Killer Cells, NK Cell Recognition, TGF-ß and Epidemiology.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 19 words, with their numbers
  1. Autoimmunity322 topics
  2. Genetic Mutations291 topic
  3. Primary Immunodeficiency291 topic
  4. Foxp3241 topic
  5. NK Cell Recognition241 topic
  6. Natural Killer Cells241 topic
  7. TGF-ß241 topic
  8. Epidemiology172 topics
  9. Antigen Presentation152 topics
  10. Dendritic Cells91 topic
  11. Immunity91 topic
  12. Mycobacterium avium91 topic
  13. Nontuberculous Mycobacteria91 topic
  14. Genetics81 topic
  15. Type 1 Diabetes81 topic
  16. Epitope Prediction61 topic
  17. Pattern Recognition Receptors61 topic
  18. Peptide-MHC Binding61 topic
  19. Toll-like Receptors61 topic

Where does Masato Ogishi work?

Masato Ogishi's main affiliation in the publication record is Rockefeller University, United States.

How many publications and citations does Masato Ogishi have?

OpenAlex counts 88 works and 8,396 citations for Masato Ogishi, an h-index of 29, ranking #66,929 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

Ask a question or share a note about Masato Ogishi. Keep it to the research: no personal attacks, advertising or links. Any reader can report a post, which collapses it for everyone until it is opened.