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Researcher · Iğdır Üniversitesi · Türkiye

Namık Kılınç

Namık Kılınç publishes mostly in Molecular Biology, Organic Chemistry and Pharmacology, on topics such as Enzyme function and inhibition, Cholinesterase and Neurodegenerative Diseases and Aldose Reductase and Taurine.

ORCID 0000-0002-9102-1370 OpenAlex
World rank
#1,164,655
of 7,710,434 ranked researchers
Rank in Türkiye
#4,302
of 117,622
Works
51
Citations
625
Citations per work
12.3
h-index
16
i10-index 18Top 46% in Molecular Biology
Namık Kılınç

What does Namık Kılınç research?

Namık Kılınç's work is mostly in Biochemistry, Genetics and Molecular Biology (29%), Medicine (18%), Chemistry (15%).

Everything else: 17 works · 32.8% of their workLife Sciences: 15 works · 29.4% of their workBiochemistry, Genetics and Molecular Biology: 15 works · 29.4% of their workMolecular Biology: 12 works · 23.8% of their workCell Biology: 3 works · 5.6% of their workPhysical Sciences: 10 works · 19.6% of their workChemistry: 8 works · 15.4% of their workOrganic Chemistry: 8 works · 15.4% of their workComputer Science: 2 works · 4.2% of their workComputational Theory and Mathematics: 2 works · 4.2% of their workHealth Sciences: 9 works · 18.2% of their workMedicine: 9 works · 18.2% of their workPharmacology: 7 works · 14.7% of their workEndocrinology, Diabetes and Metabolism: 2 works · 3.5% of their workTHEIR WORKNamık Kılınç51works
Life Sciences29%Physical Sciences20%Health Sciences18%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
  1. Everything else1732.8%
    1. Biochemistry, Genetics and Molecular Biology1529.4%
    1. Chemistry815.4%
    2. Computer Science24.2%
    1. Medicine918.2%
  1. 1Molecular Biology24% of their works
  2. 2Organic Chemistry15% of their works
  3. 3Pharmacology15% of their works
  4. 4Cell Biology6% of their works
  5. 5Computational Theory and Mathematics4% of their works
  6. 6Endocrinology, Diabetes and Metabolism4% of their works

Research topics

Which keywords describe Namık Kılınç's research?

The keywords of their largest research topics: Anticancer Agents, Pharmacology, Toxicology, Carbonic Anhydrases, Inhibitors, Acetylcholinesterase, Cholinesterase Inhibitors and Neuroprotection.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 22 words, with their numbers
  1. Anticancer Agents263 topics
  2. Pharmacology202 topics
  3. Toxicology202 topics
  4. Carbonic Anhydrases171 topic
  5. Inhibitors171 topic
  6. Acetylcholinesterase161 topic
  7. Cholinesterase Inhibitors161 topic
  8. Neuroprotection122 topics
  9. Oxidative Stress122 topics
  10. Medicinal Chemistry112 topics
  11. Aldose Reductase81 topic
  12. Taurine81 topic
  13. Molecular Docking61 topic
  14. Virtual Screening61 topic
  15. Heterocyclic Compounds51 topic
  16. Tubulin Inhibitors51 topic
  17. Antioxidant41 topic
  18. Cannabinoid Receptors41 topic
  19. Cytotoxicity41 topic
  20. Endocannabinoid System41 topic
  21. Phenolic Compounds41 topic
  22. Quinones41 topic

Where does Namık Kılınç work?

Namık Kılınç's main affiliation in the publication record is Iğdır Üniversitesi, Türkiye.

How many publications and citations does Namık Kılınç have?

OpenAlex counts 51 works and 625 citations for Namık Kılınç, an h-index of 16, ranking #1,164,655 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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