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Researcher · The University of Melbourne · Australia

Norman Ruthven Saunders

Norman Ruthven Saunders publishes mostly in Pediatrics, Perinatology and Child Health, Neurology and Molecular Biology, on topics such as Barrier Structure and Function Studies, Neonatal and fetal brain pathology and Drug Transport and Resistance Mechanisms.

ORCID 0000-0001-6660-7639 OpenAlex
World rank
#597,381
of 7,710,434 ranked researchers
Rank in Australia
#20,480
of 140,210
Works
444
Citations
14k
Citations per work
30.6
h-index
67
i10-index 204Top 0.7% in Pediatrics, Perinatology and Child Health
Norman Ruthven Saunders

What does Norman Ruthven Saunders research?

Norman Ruthven Saunders's work is mostly in Medicine (21%), Neuroscience (16%), Biochemistry, Genetics and Molecular Biology (9%).

Everything else: 239 works · 53.9% of their workLife Sciences: 112 works · 25.2% of their workNeuroscience: 71 works · 15.9% of their workNeurology: 42 works · 9.5% of their workCellular and Molecular Neuroscience: 28 works · 6.4% of their workBiochemistry, Genetics and Molecular Biology: 41 works · 9.3% of their workMolecular Biology: 41 works · 9.3% of their workHealth Sciences: 93 works · 20.9% of their workMedicine: 93 works · 20.9% of their workPediatrics, Perinatology and Child Health: 56 works · 12.5% of their workOncology: 19 works · 4.2% of their workPhysiology: 19 works · 4.2% of their workTHEIR WORKNorman RuthvenSaunders444works
Life Sciences25%Health Sciences21%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
  1. Everything else23953.9%
    1. Neuroscience7115.9%
    2. Biochemistry, Genetics and Molecular Biology419.3%
    1. Medicine9320.9%
  1. 1Pediatrics, Perinatology and Child Health12% of their works
  2. 2Neurology10% of their works
  3. 3Molecular Biology9% of their works
  4. 4Cellular and Molecular Neuroscience6% of their works
  5. 5Oncology4% of their works
  6. 6Physiology4% of their works

Research topics

Which keywords describe Norman Ruthven Saunders's research?

The keywords of their largest research topics: Inflammation, Blood-Brain Barrier, Neurovascular Unit, Hypoxic-Ischemic Encephalopathy, Neonatal Encephalopathy, Spinal Cord Injury, Synaptic Plasticity and ATP-dependent Transporters.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 19 words, with their numbers
  1. Inflammation742 topics
  2. Blood-Brain Barrier731 topic
  3. Neurovascular Unit731 topic
  4. Hypoxic-Ischemic Encephalopathy491 topic
  5. Neonatal Encephalopathy491 topic
  6. Spinal Cord Injury442 topics
  7. Synaptic Plasticity422 topics
  8. ATP-dependent Transporters401 topic
  9. Multidrug Resistance401 topic
  10. Adult Stem Cells271 topic
  11. Neurogenesis271 topic
  12. Epidemiology251 topic
  13. Neurological Classification251 topic
  14. GABAergic231 topic
  15. Neurotransmission231 topic
  16. Antiepileptic Drugs191 topic
  17. BDNF191 topic
  18. Neurotrophins191 topic
  19. Prenatal Exposure191 topic

Where does Norman Ruthven Saunders work?

Norman Ruthven Saunders's main affiliation in the publication record is The University of Melbourne, Australia.

How many publications and citations does Norman Ruthven Saunders have?

OpenAlex counts 444 works and 13,577 citations for Norman Ruthven Saunders, an h-index of 67, ranking #597,381 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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