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Researcher · Medical University of Białystok · Poland

Otylia Kowal‐Bielecka

Otylia Kowal‐Bielecka publishes mostly in Pathology and Forensic Medicine, Pulmonary and Respiratory Medicine and Rheumatology, on topics such as Systemic Sclerosis and Related Diseases, Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis and Inflammatory Myopathies and Dermatomyositis.

ORCID 0000-0002-0613-1306 OpenAlex
World rank
#703,544
of 7,710,434 ranked researchers
Rank in Poland
#4,456
of 100,960
Works
206
Citations
15k
Citations per work
71.5
h-index
38
i10-index 80Top 8.6% in Pathology and Forensic Medicine
Otylia Kowal‐Bielecka

What does Otylia Kowal‐Bielecka research?

Otylia Kowal‐Bielecka's work is mostly in Medicine (64%), Biochemistry, Genetics and Molecular Biology (6%), Immunology and Microbiology (4%).

Health Sciences: 133 works · 64.5% of their workMedicine: 133 works · 64.5% of their workPathology and Forensic Medicine: 67 works · 32.6% of their workPulmonary and Respiratory Medicine: 39 works · 18.9% of their workRheumatology: 17 works · 8.1% of their workEpidemiology: 10 works · 4.9% of their workEverything else: 54 works · 26.1% of their workLife Sciences: 19 works · 9.4% of their workBiochemistry, Genetics and Molecular Biology: 12 works · 5.7% of their workMolecular Biology: 12 works · 5.7% of their workImmunology and Microbiology: 8 works · 3.7% of their workImmunology: 8 works · 3.7% of their workTHEIR WORKOtyliaKowal‐Bielecka206works
Life Sciences9%Health Sciences64%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Medicine13364.5%
  2. Everything else5426.1%
    1. Biochemistry, Genetics and Molecular Biology125.7%
    2. Immunology and Microbiology83.7%
  1. 1Pathology and Forensic Medicine33% of their works
  2. 2Pulmonary and Respiratory Medicine19% of their works
  3. 3Rheumatology8% of their works
  4. 4Molecular Biology6% of their works
  5. 5Epidemiology5% of their works
  6. 6Immunology4% of their works

Research topics

Which keywords describe Otylia Kowal‐Bielecka's research?

The keywords of their largest research topics: Fibrosis, Autoantibodies, Treatment, Endothelial Dysfunction, TGF-ß Signaling, Pulmonary Involvement, Systemic Sclerosis and Interstitial Lung Disease.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 22 words, with their numbers
  1. Fibrosis2273 topics
  2. Autoantibodies1934 topics
  3. Treatment1672 topics
  4. Endothelial Dysfunction1662 topics
  5. TGF-ß Signaling1642 topics
  6. Pulmonary Involvement1441 topic
  7. Systemic Sclerosis1441 topic
  8. Interstitial Lung Disease862 topics
  9. Idiopathic Pulmonary Fibrosis631 topic
  10. TGF-ß631 topic
  11. Dermatomyositis231 topic
  12. Inflammatory Myopathies231 topic
  13. Pulmonary Hypertension221 topic
  14. Right Heart Assessment221 topic
  15. CCN Family201 topic
  16. Connective Tissue Growth Factor201 topic
  17. Diagnostic Criteria161 topic
  18. Inflammation161 topic
  19. Multiple Sclerosis161 topic
  20. Rheumatoid Arthritis161 topic
  21. Antiphospholipid Syndrome101 topic
  22. Systemic Lupus Erythematosus101 topic

Where does Otylia Kowal‐Bielecka work?

Otylia Kowal‐Bielecka's main affiliation in the publication record is Medical University of Białystok, Poland.

How many publications and citations does Otylia Kowal‐Bielecka have?

OpenAlex counts 206 works and 14,733 citations for Otylia Kowal‐Bielecka, an h-index of 38, ranking #703,544 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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