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Researcher · Gladstone Institutes · United States

Victoria A. Rafalski

Victoria A. Rafalski publishes mostly in Molecular Biology, Neurology and Physiology, on topics such as Neuroinflammation and Neurodegeneration Mechanisms, Neurogenesis and neuroplasticity mechanisms and Alzheimer's disease research and treatments.

OpenAlex
World rank
#928,518
of 7,710,434 ranked researchers
Rank in United States
#305,404
of 1,627,098
Works
24
Citations
2.8k
Citations per work
116.0
h-index
16
i10-index 16Top 46% in Molecular Biology
Victoria A. Rafalski

What does Victoria A. Rafalski research?

Victoria A. Rafalski's work is mostly in Neuroscience (24%), Biochemistry, Genetics and Molecular Biology (18%), Medicine (16%).

Life Sciences: 12 works · 49.3% of their workNeuroscience: 6 works · 23.9% of their workNeurology: 4 works · 17.9% of their workDevelopmental Neuroscience: 1 works · 6.0% of their workBiochemistry, Genetics and Molecular Biology: 4 works · 17.9% of their workMolecular Biology: 4 works · 17.9% of their workImmunology and Microbiology: 2 works · 7.5% of their workImmunology: 2 works · 7.5% of their workEverything else: 8 works · 34.3% of their workHealth Sciences: 4 works · 16.4% of their workMedicine: 4 works · 16.4% of their workPhysiology: 2 works · 10.4% of their workNeurology: 1 works · 6.0% of their workTHEIR WORKVictoria A.Rafalski24works
Life Sciences49%Health Sciences16%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Neuroscience623.9%
    2. Biochemistry, Genetics and Molecular Biology417.9%
    3. Immunology and Microbiology27.5%
  2. Everything else834.3%
    1. Medicine416.4%
  1. 1Molecular Biology18% of their works
  2. 2Neurology18% of their works
  3. 3Physiology10% of their works
  4. 4Immunology8% of their works
  5. 5Developmental Neuroscience6% of their works
  6. 6Neurology6% of their works

Research topics

Which keywords describe Victoria A. Rafalski's research?

The keywords of their largest research topics: Neurodegeneration, Brain Development, Neuroinflammation, Innate Immunity, Oxidative Stress, Astrocytes, Microglia and Aging.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 25 words, with their numbers
  1. Neurodegeneration143 topics
  2. Brain Development122 topics
  3. Neuroinflammation122 topics
  4. Innate Immunity102 topics
  5. Oxidative Stress93 topics
  6. Astrocytes81 topic
  7. Microglia81 topic
  8. Aging52 topics
  9. Insulin Signaling52 topics
  10. Adult Stem Cells41 topic
  11. Amyloid Hypothesis41 topic
  12. Cancer42 topics
  13. Neurogenesis41 topic
  14. Regulation42 topics
  15. Tau Pathology41 topic
  16. Genetic Regulation31 topic
  17. Induced Pluripotent Stem Cells31 topic
  18. Longevity31 topic
  19. Pluripotent Stem Cells31 topic
  20. FoxO Transcription Factors21 topic
  21. Gene Expression21 topic
  22. Inflammation21 topic
  23. MicroRNA21 topic
  24. S100 Proteins21 topic
  25. cell cycle regulation21 topic

Where does Victoria A. Rafalski work?

Victoria A. Rafalski's main affiliation in the publication record is Gladstone Institutes, United States.

How many publications and citations does Victoria A. Rafalski have?

OpenAlex counts 24 works and 2,785 citations for Victoria A. Rafalski, an h-index of 16, ranking #928,518 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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