Science Explorer Back to mapMap
Discussion
Researcher · Kyoto Prefectural University · Japan

Yasutomi Kamei

Yasutomi Kamei publishes mostly in Molecular Biology, Physiology and Epidemiology, on topics such as Adipose Tissue and Metabolism, Muscle Physiology and Disorders and Adipokines, Inflammation, and Metabolic Diseases.

ORCID 0000-0003-2778-926X OpenAlex
World rank
#272,188
of 7,710,434 ranked researchers
Rank in Japan
#2,248
of 383,136
Works
152
Citations
13k
Citations per work
86.3
h-index
49
i10-index 80Top 3.9% in Molecular Biology
Yasutomi Kamei

What does Yasutomi Kamei research?

Yasutomi Kamei's work is mostly in Biochemistry, Genetics and Molecular Biology (42%), Medicine (30%).

Life Sciences: 63 works · 41.5% of their workBiochemistry, Genetics and Molecular Biology: 63 works · 41.5% of their workMolecular Biology: 49 works · 32.2% of their workCell Biology: 7 works · 4.9% of their workGenetics: 7 works · 4.4% of their workHealth Sciences: 45 works · 29.9% of their workMedicine: 45 works · 29.9% of their workPhysiology: 32 works · 21.3% of their workEpidemiology: 9 works · 5.6% of their workSurgery: 5 works · 3.0% of their workEverything else: 43 works · 28.6% of their workTHEIR WORKYasutomi Kamei152works
Life Sciences42%Health Sciences30%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology6341.5%
    1. Medicine4529.9%
  3. Everything else4328.6%
  1. 1Molecular Biology32% of their works
  2. 2Physiology21% of their works
  3. 3Epidemiology6% of their works
  4. 4Cell Biology5% of their works
  5. 5Genetics4% of their works
  6. 6Surgery3% of their works

Research topics

Which keywords describe Yasutomi Kamei's research?

The keywords of their largest research topics: Insulin Resistance, Exercise, Brown Adipose Tissue, Thermogenesis, FoxO Transcription Factors, Metabolism, Aging and Muscle Regeneration.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 26 words, with their numbers
  1. Insulin Resistance962 topics
  2. Exercise922 topics
  3. Brown Adipose Tissue741 topic
  4. Thermogenesis741 topic
  5. FoxO Transcription Factors562 topics
  6. Metabolism483 topics
  7. Aging433 topics
  8. Muscle Regeneration411 topic
  9. Skeletal Muscle Atrophy411 topic
  10. Inflammation372 topics
  11. Obesity372 topics
  12. Nuclear Receptors262 topics
  13. Insulin Signaling252 topics
  14. Oxidative Stress252 topics
  15. Adipose Tissue221 topic
  16. Cardiovascular Disease221 topic
  17. Nutrition181 topic
  18. Protein181 topic
  19. Apoptosis151 topic
  20. PPARs151 topic
  21. Peroxisome Proliferator-Activated Receptors151 topic
  22. cell cycle regulation151 topic
  23. Acute Promyelocytic Leukemia111 topic
  24. Retinoic Acid111 topic
  25. Genetic Regulation101 topic
  26. Longevity101 topic

Where does Yasutomi Kamei work?

Yasutomi Kamei's main affiliation in the publication record is Kyoto Prefectural University, Japan.

How many publications and citations does Yasutomi Kamei have?

OpenAlex counts 152 works and 13,123 citations for Yasutomi Kamei, an h-index of 49, ranking #272,188 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

Ask a question or share a note about Yasutomi Kamei. Keep it to the research: no personal attacks, advertising or links. Any reader can report a post, which collapses it for everyone until it is opened.