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Researcher · PDX Pharmaceuticals (United States) · United States

Moataz M. Reda

Moataz M. Reda publishes mostly in Molecular Biology, Biomedical Engineering and Oncology, on topics such as Nanoplatforms for cancer theranostics, RNA Interference and Gene Delivery and Immunotherapy and Immune Responses.

OpenAlex
World rank
#1,523,211
of 7,710,434 ranked researchers
Rank in United States
#476,518
of 1,627,098
Works
37
Citations
975
Citations per work
26.4
h-index
14
i10-index 15Top 55% in Molecular Biology
Moataz M. Reda

What does Moataz M. Reda research?

Moataz M. Reda's work is mostly in Biochemistry, Genetics and Molecular Biology (29%), Engineering (19%), Medicine (16%).

Life Sciences: 16 works · 42.2% of their workBiochemistry, Genetics and Molecular Biology: 11 works · 28.9% of their workMolecular Biology: 8 works · 22.2% of their workBiotechnology: 2 works · 6.7% of their workImmunology and Microbiology: 5 works · 13.3% of their workImmunology: 5 works · 13.3% of their workPhysical Sciences: 10 works · 26.7% of their workEngineering: 7 works · 18.9% of their workBiomedical Engineering: 7 works · 18.9% of their workMaterials Science: 3 works · 7.8% of their workBiomaterials: 3 works · 7.8% of their workHealth Sciences: 6 works · 15.6% of their workMedicine: 6 works · 15.6% of their workOncology: 6 works · 15.6% of their workEverything else: 6 works · 15.5% of their workTHEIR WORKMoataz M. Reda37works
Life Sciences42%Physical Sciences27%Health Sciences16%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology1128.9%
    2. Immunology and Microbiology513.3%
    1. Engineering718.9%
    2. Materials Science37.8%
    1. Medicine615.6%
  4. Everything else615.5%
  1. 1Molecular Biology22% of their works
  2. 2Biomedical Engineering19% of their works
  3. 3Oncology16% of their works
  4. 4Immunology13% of their works
  5. 5Biomaterials8% of their works
  6. 6Biotechnology7% of their works

Research topics

Which keywords describe Moataz M. Reda's research?

The keywords of their largest research topics: Tumor Targeting, Nanoparticles, Immunotherapy, Gene Therapy, Photothermal Therapy, Reactive Oxygen Species, Innate Immunity and RNA Interference.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 21 words, with their numbers
  1. Tumor Targeting273 topics
  2. Nanoparticles263 topics
  3. Immunotherapy202 topics
  4. Gene Therapy152 topics
  5. Photothermal Therapy141 topic
  6. Reactive Oxygen Species141 topic
  7. Innate Immunity112 topics
  8. RNA Interference91 topic
  9. siRNA Delivery91 topic
  10. Dendritic Cells81 topic
  11. Immune Checkpoint Blockade81 topic
  12. Immunity81 topic
  13. PD-1 and PD-L181 topic
  14. Drug Delivery71 topic
  15. Nanocarriers71 topic
  16. Arginine Deprivation61 topic
  17. Bacterial Therapy61 topic
  18. DNA nanotechnology51 topic
  19. Self-Assembly51 topic
  20. Interferon Signaling31 topic
  21. Viral Infection31 topic

Where does Moataz M. Reda work?

Moataz M. Reda's main affiliation in the publication record is PDX Pharmaceuticals (United States), United States.

How many publications and citations does Moataz M. Reda have?

OpenAlex counts 37 works and 975 citations for Moataz M. Reda, an h-index of 14, ranking #1,523,211 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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