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Researcher · Bicycle Therapeutics (United Kingdom) · United Kingdom

Philip Huxley

Philip Huxley publishes mostly in Molecular Biology, Cellular and Molecular Neuroscience and Oncology, on topics such as Axon Guidance and Neuronal Signaling, Peptidase Inhibition and Analysis and Monoclonal and Polyclonal Antibodies Research.

OpenAlex
World rank
#1,511,092
of 7,710,434 ranked researchers
Rank in United Kingdom
#115,970
of 367,258
Works
36
Citations
772
Citations per work
21.4
h-index
12
i10-index 12Top 65% in Molecular Biology
Philip Huxley

What does Philip Huxley research?

Philip Huxley's work is mostly in Biochemistry, Genetics and Molecular Biology (24%), Medicine (21%), Neuroscience (13%).

Life Sciences: 13 works · 36.5% of their workBiochemistry, Genetics and Molecular Biology: 9 works · 23.8% of their workMolecular Biology: 6 works · 15.9% of their workCancer Research: 3 works · 7.9% of their workNeuroscience: 5 works · 12.7% of their workCellular and Molecular Neuroscience: 5 works · 12.7% of their workEverything else: 13 works · 35.0% of their workHealth Sciences: 7 works · 20.6% of their workMedicine: 7 works · 20.6% of their workOncology: 4 works · 11.1% of their workRadiology, Nuclear Medicine and Imaging: 3 works · 9.5% of their workPhysical Sciences: 3 works · 7.9% of their workChemistry: 3 works · 7.9% of their workOrganic Chemistry: 3 works · 7.9% of their workTHEIR WORKPhilip Huxley36works
Life Sciences36%Physical Sciences8%Health Sciences21%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology923.8%
    2. Neuroscience512.7%
  2. Everything else1335.0%
    1. Medicine720.6%
    1. Chemistry37.9%
  1. 1Molecular Biology16% of their works
  2. 2Cellular and Molecular Neuroscience13% of their works
  3. 3Oncology11% of their works
  4. 4Radiology, Nuclear Medicine and Imaging10% of their works
  5. 5Organic Chemistry8% of their works
  6. 6Cancer Research8% of their works

Research topics

Which keywords describe Philip Huxley's research?

The keywords of their largest research topics: Cell Signaling, Angiogenesis, Eph receptors, Metastasis, Semaphorins, Fc Receptors, Fibroblast Activation Protein and Therapeutic Antibodies.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 19 words, with their numbers
  1. Cell Signaling92 topics
  2. Angiogenesis82 topics
  3. Eph receptors61 topic
  4. Metastasis62 topics
  5. Semaphorins61 topic
  6. Fc Receptors51 topic
  7. Fibroblast Activation Protein51 topic
  8. Therapeutic Antibodies51 topic
  9. Tumor Microenvironment51 topic
  10. Bacterial Therapy31 topic
  11. Inflammation31 topic
  12. Matrix Metalloproteinases31 topic
  13. S100 Proteins31 topic
  14. Tissue Inhibitors of Metalloproteinases31 topic
  15. Tumor Targeting31 topic
  16. Antibiotic Resistance21 topic
  17. Ecteinascidin21 topic
  18. Multidrug-Resistant Bacteria21 topic
  19. Tetrahydroisoquinoline21 topic

Where does Philip Huxley work?

Philip Huxley's main affiliation in the publication record is Bicycle Therapeutics (United Kingdom), United Kingdom.

How many publications and citations does Philip Huxley have?

OpenAlex counts 36 works and 772 citations for Philip Huxley, an h-index of 12, ranking #1,511,092 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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