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Researcher · Washington University in St. Louis · United States

Robert D. Schreiber

Robert D. Schreiber publishes mostly in Immunology, Oncology and Molecular Biology, on topics such as Immunotherapy and Immune Responses, Immune Cell Function and Interaction and Cytokine Signaling Pathways and Interactions.

ORCID 0000-0003-1590-2341 OpenAlex
World rank
#9,340
of 7,710,434 ranked researchers
Rank in United States
#3,203
of 1,627,098
Works
710
Citations
111k
Citations per work
156.3
h-index
134
i10-index 335Top 0.1% in Immunology
Robert D. Schreiber

What does Robert D. Schreiber research?

Robert D. Schreiber's work is mostly in Immunology and Microbiology (35%), Medicine (21%), Biochemistry, Genetics and Molecular Biology (8%).

Life Sciences: 333 works · 46.9% of their workImmunology and Microbiology: 246 works · 34.6% of their workImmunology: 246 works · 34.6% of their workBiochemistry, Genetics and Molecular Biology: 60 works · 8.5% of their workMolecular Biology: 60 works · 8.5% of their workNeuroscience: 27 works · 3.8% of their workCellular and Molecular Neuroscience: 27 works · 3.8% of their workEverything else: 228 works · 32.1% of their workHealth Sciences: 149 works · 21.0% of their workMedicine: 149 works · 21.0% of their workOncology: 98 works · 13.8% of their workRadiology, Nuclear Medicine and Imaging: 35 works · 4.9% of their workHematology: 16 works · 2.3% of their workTHEIR WORKRobert D.Schreiber710works
Life Sciences47%Health Sciences21%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Immunology and Microbiology24634.6%
    2. Biochemistry, Genetics and Molecular Biology608.5%
    3. Neuroscience273.8%
  2. Everything else22832.1%
    1. Medicine14921.0%
  1. 1Immunology35% of their works
  2. 2Oncology14% of their works
  3. 3Molecular Biology8% of their works
  4. 4Radiology, Nuclear Medicine and Imaging5% of their works
  5. 5Cellular and Molecular Neuroscience4% of their works
  6. 6Hematology2% of their works

Research topics

Which keywords describe Robert D. Schreiber's research?

The keywords of their largest research topics: Innate Immunity, Inflammation, Tumor Microenvironment, Dendritic Cells, Immunity, Pattern Recognition Receptors, Natural Killer Cells and NK Cell Recognition.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 20 words, with their numbers
  1. Innate Immunity1752 topics
  2. Inflammation1462 topics
  3. Tumor Microenvironment1402 topics
  4. Dendritic Cells1231 topic
  5. Immunity1231 topic
  6. Pattern Recognition Receptors1232 topics
  7. NK Cell Recognition1031 topic
  8. Natural Killer Cells1031 topic
  9. IL-6751 topic
  10. STAT3751 topic
  11. Innate Antiviral Responses711 topic
  12. Toll-like Receptors711 topic
  13. Fc Receptors671 topic
  14. Therapeutic Antibodies671 topic
  15. Immune Checkpoint Blockade651 topic
  16. PD-1 and PD-L1651 topic
  17. Foxp3551 topic
  18. TGF-ß551 topic
  19. Interferon Signaling521 topic
  20. Viral Infection521 topic

Where does Robert D. Schreiber work?

Robert D. Schreiber's main affiliation in the publication record is Washington University in St. Louis, United States.

How many publications and citations does Robert D. Schreiber have?

OpenAlex counts 710 works and 110,947 citations for Robert D. Schreiber, an h-index of 134, ranking #9,340 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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