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Researcher · Max Planck Institute of Biochemistry · Germany

F. Ulrich Hartl

F. Ulrich Hartl publishes mostly in Molecular Biology, Materials Chemistry and Cell Biology, on topics such as Heat shock proteins research, Protein Structure and Dynamics and Enzyme Structure and Function.

ORCID 0000-0002-7941-135X OpenAlex
World rank
#3,965
of 7,710,434 ranked researchers
Rank in Germany
#147
of 364,343
Works
494
Citations
71k
Citations per work
143.2
h-index
136
i10-index 288Top 0.1% in Molecular Biology
F. Ulrich Hartl

What does F. Ulrich Hartl research?

F. Ulrich Hartl's work is mostly in Biochemistry, Genetics and Molecular Biology (65%), Materials Science (10%), Immunology and Microbiology (4%).

Life Sciences: 359 works · 72.7% of their workBiochemistry, Genetics and Molecular Biology: 321 works · 64.9% of their workMolecular Biology: 276 works · 55.9% of their workCell Biology: 32 works · 6.5% of their workGenetics: 12 works · 2.5% of their workImmunology and Microbiology: 20 works · 4.1% of their workImmunology: 20 works · 4.1% of their workNeuroscience: 18 works · 3.7% of their workCellular and Molecular Neuroscience: 18 works · 3.7% of their workEverything else: 85 works · 17.2% of their workPhysical Sciences: 50 works · 10.1% of their workMaterials Science: 50 works · 10.1% of their workMaterials Chemistry: 50 works · 10.1% of their workTHEIR WORKF. Ulrich Hartl494works
Life Sciences73%Physical Sciences10%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology32164.9%
    2. Immunology and Microbiology204.1%
    3. Neuroscience183.7%
  2. Everything else8517.2%
    1. Materials Science5010.1%
  1. 1Molecular Biology56% of their works
  2. 2Materials Chemistry10% of their works
  3. 3Cell Biology6% of their works
  4. 4Immunology4% of their works
  5. 5Cellular and Molecular Neuroscience4% of their works
  6. 6Genetics2% of their works

Research topics

Which keywords describe F. Ulrich Hartl's research?

The keywords of their largest research topics: Protein Folding, Cancer Therapy, Molecular Chaperones, Proteostasis, Protein, Structure, Cell Death and Oxidative Stress.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 22 words, with their numbers
  1. Protein Folding2522 topics
  2. Cancer Therapy2342 topics
  3. Molecular Chaperones1901 topic
  4. Proteostasis1901 topic
  5. Protein1231 topic
  6. Structure1231 topic
  7. Cell Death1192 topics
  8. Oxidative Stress1192 topics
  9. Macromolecular Crystallography1151 topic
  10. Structure Solution1151 topic
  11. Chaperones982 topics
  12. Mitochondrial Dysfunction932 topics
  13. Ribosome651 topic
  14. Translation651 topic
  15. Endoplasmic Reticulum621 topic
  16. Stress621 topic
  17. Mitochondria571 topic
  18. Reactive Oxygen Species571 topic
  19. Immunotoxin441 topic
  20. Ricin Poisoning441 topic
  21. Huntington's Disease361 topic
  22. Protein Aggregation361 topic

Where does F. Ulrich Hartl work?

F. Ulrich Hartl's main affiliation in the publication record is Max Planck Institute of Biochemistry, Germany.

How many publications and citations does F. Ulrich Hartl have?

OpenAlex counts 494 works and 70,764 citations for F. Ulrich Hartl, an h-index of 136, ranking #3,965 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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