Science Explorer Back to mapMap
Discussion
Researcher · Olgahospital · Germany

Günther E Dannecker

Günther E Dannecker publishes mostly in Immunology, Rheumatology and Surgery, on topics such as T-cell and B-cell Immunology, Immune Cell Function and Interaction and Autoimmune and Inflammatory Disorders Research.

OpenAlex
World rank
#7,442,492
of 7,710,434 ranked researchers
Rank in Germany
#356,316
of 364,343
Works
100
Citations
727
Citations per work
7.3
h-index
17
i10-index 28Top 40% in Immunology
Günther E Dannecker

What does Günther E Dannecker research?

Günther E Dannecker's work is mostly in Immunology and Microbiology (34%), Medicine (24%), Biochemistry, Genetics and Molecular Biology (5%).

Life Sciences: 40 works · 39.5% of their workImmunology and Microbiology: 34 works · 34.5% of their workImmunology: 34 works · 34.5% of their workBiochemistry, Genetics and Molecular Biology: 5 works · 5.0% of their workMolecular Biology: 5 works · 5.0% of their workEverything else: 36 works · 36.0% of their workHealth Sciences: 24 works · 24.5% of their workMedicine: 24 works · 24.5% of their workRheumatology: 10 works · 10.0% of their workSurgery: 6 works · 6.0% of their workHematology: 5 works · 5.3% of their workEpidemiology: 3 works · 3.2% of their workTHEIR WORKGünther EDannecker100works
Life Sciences40%Health Sciences24%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Immunology and Microbiology3434.5%
    2. Biochemistry, Genetics and Molecular Biology55.0%
  2. Everything else3636.0%
    1. Medicine2424.5%
  1. 1Immunology34% of their works
  2. 2Rheumatology10% of their works
  3. 3Surgery6% of their works
  4. 4Hematology5% of their works
  5. 5Molecular Biology5% of their works
  6. 6Epidemiology3% of their works

Research topics

Which keywords describe Günther E Dannecker's research?

The keywords of their largest research topics: Foxp3, TGF-ß, Natural Killer Cells, NK Cell Recognition, Toll-like Receptors, Inflammation, Hemophagocytic Lymphohistiocytosis and Juvenile Idiopathic Arthritis.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 18 words, with their numbers
  1. Foxp3301 topic
  2. TGF-ß301 topic
  3. NK Cell Recognition281 topic
  4. Natural Killer Cells281 topic
  5. Toll-like Receptors202 topics
  6. Inflammation162 topics
  7. Hemophagocytic Lymphohistiocytosis141 topic
  8. Juvenile Idiopathic Arthritis141 topic
  9. Dendritic Cells131 topic
  10. Immunity131 topic
  11. Antiphospholipid Syndrome121 topic
  12. Systemic Lupus Erythematosus121 topic
  13. Genetic Mutations91 topic
  14. Primary Immunodeficiency91 topic
  15. Inflammasome81 topic
  16. Innate Antiviral Responses81 topic
  17. NLRP381 topic
  18. Pattern Recognition Receptors81 topic

Where does Günther E Dannecker work?

Günther E Dannecker's main affiliation in the publication record is Olgahospital, Germany.

How many publications and citations does Günther E Dannecker have?

OpenAlex counts 100 works and 727 citations for Günther E Dannecker, an h-index of 17, ranking #7,442,492 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

Ask a question or share a note about Günther E Dannecker. Keep it to the research: no personal attacks, advertising or links. Any reader can report a post, which collapses it for everyone until it is opened.