Science Explorer Back to mapMap
Discussion
Researcher · Central Institute for Experimental Animals · Japan

Motohito Goto

Motohito Goto publishes mostly in Immunology, Molecular Biology and Genetics, on topics such as Immune Cell Function and Interaction, T-cell and B-cell Immunology and Animal Genetics and Reproduction.

ORCID 0000-0001-6288-4583 OpenAlex
World rank
#2,526,031
of 7,710,434 ranked researchers
Rank in Japan
#51,418
of 383,136
Works
67
Citations
1.8k
Citations per work
26.4
h-index
22
i10-index 35Top 26% in Immunology
Motohito Goto

What does Motohito Goto research?

Motohito Goto's work is mostly in Biochemistry, Genetics and Molecular Biology (26%), Immunology and Microbiology (25%), Medicine (9%).

Life Sciences: 36 works · 54.4% of their workBiochemistry, Genetics and Molecular Biology: 17 works · 25.9% of their workMolecular Biology: 12 works · 18.3% of their workGenetics: 5 works · 7.6% of their workImmunology and Microbiology: 17 works · 24.9% of their workImmunology: 17 works · 24.9% of their workNeuroscience: 2 works · 3.6% of their workCellular and Molecular Neuroscience: 2 works · 3.6% of their workEverything else: 24 works · 36.4% of their workHealth Sciences: 6 works · 9.2% of their workMedicine: 6 works · 9.2% of their workOncology: 3 works · 5.1% of their workPhysiology: 3 works · 4.1% of their workTHEIR WORKMotohito Goto67works
Life Sciences54%Health Sciences9%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology1725.9%
    2. Immunology and Microbiology1724.9%
    3. Neuroscience23.6%
  2. Everything else2436.4%
    1. Medicine69.2%
  1. 1Immunology25% of their works
  2. 2Molecular Biology18% of their works
  3. 3Genetics8% of their works
  4. 4Oncology5% of their works
  5. 5Physiology4% of their works
  6. 6Cellular and Molecular Neuroscience4% of their works

Research topics

Which keywords describe Motohito Goto's research?

The keywords of their largest research topics: Embryonic Stem Cells, Natural Killer Cells, NK Cell Recognition, Foxp3, Immunity, Inflammation, TGF-ß and Genetic Modification.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 19 words, with their numbers
  1. Embryonic Stem Cells142 topics
  2. NK Cell Recognition121 topic
  3. Natural Killer Cells121 topic
  4. Foxp3101 topic
  5. Immunity102 topics
  6. Inflammation102 topics
  7. TGF-ß101 topic
  8. Genetic Modification81 topic
  9. Genome Engineering81 topic
  10. RNA-Guided81 topic
  11. Transgenic Animals81 topic
  12. Induced Pluripotent Stem Cells61 topic
  13. Pluripotent Stem Cells61 topic
  14. Activation51 topic
  15. Cancer Immunotherapy51 topic
  16. Dendritic Cells51 topic
  17. Macrophage51 topic
  18. Pattern Recognition Receptors51 topic
  19. Toll-like Receptors51 topic

Where does Motohito Goto work?

Motohito Goto's main affiliation in the publication record is Central Institute for Experimental Animals, Japan.

How many publications and citations does Motohito Goto have?

OpenAlex counts 67 works and 1,767 citations for Motohito Goto, an h-index of 22, ranking #2,526,031 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

Ask a question or share a note about Motohito Goto. Keep it to the research: no personal attacks, advertising or links. Any reader can report a post, which collapses it for everyone until it is opened.