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Researcher · University of Coimbra · Portugal

Ricardo Pires das Neves

Ricardo Pires das Neves publishes mostly in Molecular Biology, Cancer Research and Immunology, on topics such as MicroRNA in disease regulation, RNA Interference and Gene Delivery and Extracellular vesicles in disease.

ORCID 0000-0003-2291-6610 OpenAlex
World rank
#1,813,018
of 7,710,434 ranked researchers
Rank in Portugal
#7,743
of 41,760
Works
55
Citations
2.3k
Citations per work
41.1
h-index
22
i10-index 34Top 28% in Molecular Biology
Ricardo Pires das Neves

What does Ricardo Pires das Neves research?

Ricardo Pires das Neves's work is mostly in Biochemistry, Genetics and Molecular Biology (47%), Immunology and Microbiology (4%), Environmental Science (4%).

Life Sciences: 28 works · 51.4% of their workBiochemistry, Genetics and Molecular Biology: 26 works · 47.2% of their workMolecular Biology: 22 works · 39.6% of their workCancer Research: 4 works · 7.6% of their workImmunology and Microbiology: 2 works · 4.2% of their workImmunology: 2 works · 4.2% of their workEverything else: 21 works · 37.4% of their workPhysical Sciences: 4 works · 7.7% of their workEnvironmental Science: 2 works · 4.2% of their workHealth, Toxicology and Mutagenesis: 2 works · 4.2% of their workEngineering: 2 works · 3.5% of their workBiomedical Engineering: 2 works · 3.5% of their workHealth Sciences: 2 works · 3.5% of their workMedicine: 2 works · 3.5% of their workSurgery: 2 works · 3.5% of their workTHEIR WORKRicardo Pires dasNeves55works
Life Sciences51%Physical Sciences8%Health Sciences4%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology2647.2%
    2. Immunology and Microbiology24.2%
  2. Everything else2137.4%
    1. Environmental Science24.2%
    2. Engineering23.5%
    1. Medicine23.5%
  1. 1Molecular Biology40% of their works
  2. 2Cancer Research8% of their works
  3. 3Immunology4% of their works
  4. 4Health, Toxicology and Mutagenesis4% of their works
  5. 5Biomedical Engineering4% of their works
  6. 6Surgery4% of their works

Research topics

Which keywords describe Ricardo Pires das Neves's research?

The keywords of their largest research topics: Biogenesis, Cancer, Exosomes, Extracellular Vesicles, Gene Expression, MicroRNA, RNA Interference and siRNA Delivery.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 18 words, with their numbers
  1. Biogenesis142 topics
  2. Cancer142 topics
  3. Exosomes71 topic
  4. Extracellular Vesicles71 topic
  5. Gene Expression71 topic
  6. MicroRNA71 topic
  7. RNA Interference71 topic
  8. siRNA Delivery71 topic
  9. Alternative Splicing41 topic
  10. Gene Set Enrichment Analysis41 topic
  11. Protein-Protein Interaction Networks41 topic
  12. RNA-Binding Proteins41 topic
  13. Cell Mechanics31 topic
  14. DNA nanotechnology31 topic
  15. Extracellular Matrix31 topic
  16. HIF-131 topic
  17. Self-Assembly31 topic
  18. Warburg Effect31 topic

Where does Ricardo Pires das Neves work?

Ricardo Pires das Neves's main affiliation in the publication record is University of Coimbra, Portugal.

How many publications and citations does Ricardo Pires das Neves have?

OpenAlex counts 55 works and 2,262 citations for Ricardo Pires das Neves, an h-index of 22, ranking #1,813,018 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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